Activated MET acts as a salvage signal after treatment with alectinib, a selective ALK inhibitor, in ALK-positive non-small cell lung cancer
![PDF] Non-Small Cell Lung Cancer Cells Acquire Resistance to the ALK Inhibitor Alectinib by Activating Alternative Receptor Tyrosine Kinases. | Semantic Scholar](https://d3i71xaburhd42.cloudfront.net/30e359daeff4eb0d4476cac93e5de239e079bad1/9-Figure6-1.png "PDF] Non-Small Cell Lung Cancer Cells Acquire Resistance to the ALK Inhibitor Alectinib by Activating Alternative Receptor Tyrosine Kinases. | Semantic Scholar")
PDF] Non-Small Cell Lung Cancer Cells Acquire Resistance to the ALK Inhibitor Alectinib by Activating Alternative Receptor Tyrosine Kinases. | Semantic Scholar
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Crizotinib-induced antitumour activity in human alveolar rhabdomyosarcoma cells is not solely dependent on ALK and MET inhibition | Journal of Experimental & Clinical Cancer Research | Full Text
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Activated MET acts as a salvage signal after treatment with alectinib, a selective ALK inhibitor, in ALK-positive non-small ce
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MET and TP 53: How They Relate to ALK Lung Cancer — ALK POSITIVE
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